Antioxidant Protect synergistically combines many nutrients that have positive effects on the body’s detoxification process. This formula contains multiple nutrients known to raise glutathione levels, making it helpful for supporting phase II liver detoxification. It also combats free radicals and helps detoxify harmful chemicals including heavy metals. When taken with NAC, Lleucine prevents mercury from being reabsorbed into the central nervous system. Antioxidant Protect is also designed to aid the production of metallothionein. The vitamin E is 60% gamma, mixed tocopherols. Lipoic acid regenerates vitamins E and C and supplies sulfur for detoxification. This powerful formula also provides the well-researched antioxidants green tea, grape seed extract and curcumin.
|Green tea extract (Camellia sinensis-Leaf) (98% Polyphenols, 45% Epigallocatechin 3-gallate, 20:1)||25 mg|
|Turmeric (Curcuma longa-Root) (95% Curcuminoids)||25 mg|
|Alpha Lipoic Acid||45 mg|
|Manganese (Manganese bisglycinate)||1.5 mg|
|Tocopherols (Mixed tocopherols)||52.5 mg|
|Molybdenum (Molybdenum bisglycinate)||50 mg|
|Selenium (Selenomethionine)||50 mg|
|Vitamin C (Ascorbic acid)||250 mg|
|Vitamin E (d-alpha Tocopherol)||2.7 mg AT (4 IU)|
|Grape Seed Extract (Vitis vinifera-Seed) (95% Oligomeric proanthocyanidins, 50:1)||25 mg|
|Zinc (Zinc bisglycinate)||7.5 mg|
Non-Medical Ingredients:Microcrystalline cellulose, vegetable stearate, silicon dioxide.
Adults: Take two capsules per day with a meal or as directed by your health care practitioner. Take a few hours before or after taking other medications. Consult a health care practitioner for use beyond 12 weeks.
Warnings: Consult a health care practitioner prior to use if you are pregnant, breastfeeding, have diabetes, cardiovascular disease, iron deficiency, cancer, liver disorder or develop symptoms of liver trouble, or if you are taking blood thinners.
Exposure to toxic metals has become an increasingly recognized source of illness worldwide. Both cadmium and arsenic are ubiquitous in the environment, and exposure through food and water as well as occupational sources can contribute to a well-defined spectrum of disease. The symptom picture of arsenic toxicity is characterized by dermal lesions, anemia, and an increased risk for cardiovascular disease, diabetes, and liver damage. Cadmium has a significant effect on renal function, and as a result alters bone metabolism, leading to osteoporosis and osteomalacia. Cadmium-induced genotoxicity also increases risk for several cancers. The mechanisms of arsenic- and cadmium-induced damage include the production of free radicals that alter mitochondrial activity and genetic information. The metabolism and excretion of these heavy metals depend on the presence of antioxidants and thiols that aid arsenic methylation and both arsenic and cadmium metallothionein-binding. S-adenosylmethionine, lipoic acid, glutathione, selenium, zinc, N-acetylcysteine (NAC), methionine, cysteine, alpha-tocopherol, and ascorbic acid have specific roles in the mitigation of heavy metal toxicity. Several antioxidants including NAC, zinc, methionine, and cysteine, when used in conjunction with standard chelating agents, can improve the mobilization and excretion of arsenic and cadmium.
Cadmium is known to be a potent pulmonary carcinogen to human beings and to induce prostate tumor. The sequestration of cadmium, an extremely toxic element to living cells, which is performed by biological ligands such as amino acids, peptides, proteins or enzymes is important to minimize its participation in such deleterious processes. The synthesis of metallothionein is induced by a wide range of metals, in which cadmium is a particularly potent inducer. This protein is usually associated with cadmium exposure in man. Because metallothioneins may act as a detoxification agent for cadmium and chelation involves sulfur donor atoms, we administered only cadmium, cysteine, or methionine to rats and also each of these S-amino acids together with cadmium and measured the production of superoxide radicals derived from the conversion of xanthine dehydrogenase to xanthine oxidase. It could be seen in this work that the presence of cadmium enhances this conversion. However, its inoculation with cysteine or methionine almost completely diminishes this effect and this can be the result of the fact that these amino acids complex Cd(II). Thus, these compounds can be a model of the action of metallothionein, removing cadmium from circulation and preventing its deleterious effect.
The effects of some methyl-containing compounds added to a choline-deficient diet on the metallothionein mRNA level in the rat liver were studied. The addition of choline or carnitine to the choline-deficient diet did not induce a gain in body weight, while the addition of either betaine or methionine to the choline-deficient diet, or of methionine to the choline-deficient diet with choline significantly increased the body weight. The metallothionein mRNA level in the liver of rats fed on the choline-deficient diet was similar to that of rats fed on the choline-deficient diet with choline, betaine or carnitine. However, the addition of methionine to the choline-deficient diet with or without choline caused a marked suppression in the metallothionein mRNA level in the liver. It is thus surmised that the metallothionein mRNA level in the liver might be regulated by the dietary content of methionine.
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